Disturbance of pulmonary blood circulation
Contents:
Description:
Disturbances of blood circulation in system of pulmonary blood supply owing to cordial and to others of the reasons, leading to development of a pulmonary heart, pulmonary hypertensia.
Symptoms of Disturbance of pulmonary blood circulation:
Disturbances of pulmonary blood circulation owing to cardiovascular diseases, for example inborn heart diseases or states, are followed by increase in pressure in the left auricle (a mitral stenosis, etc.).
Increase in pulmonary vascular resistance is possible at all pulmonary diseases which are followed by an anoxemia as the anoxemia is often combined with an alveolar hypoxia - a powerful incentive for narrowing of pulmonary arterioles. The long and expressed alveolar hypoxia can lead to substantial permanent increase of pulmonary vascular resistance and eventually - to pulmonary hypertensia. Its risk is highest at HOZL, intersticial diseases of lungs, thorax diseases, and also hypoventilation and an apnoea in a dream at obesity. The risk of pulmonary hypertensia is even more if the disease is followed by defeat of pulmonary vessels.
At diseases of pulmonary vessels the main reason for increase in pulmonary vascular resistance - reduction of total area of cross-section of a pulmonary vascular bed. The role of an alveolar hypoxia in this case is small. At repeated TELA the cross-sectional area of a pulmonary vascular bed decreases at the expense of occlusion of branches of a pulmonary artery blood clots.
Primary pulmonary hypertensia and pulmonary hypertensia at a system scleroderma are caused by a generalized obliteration of pulmonary arterioles and small arteries.
Causes of infringement of pulmonary blood circulation:
There are two main pathophysiological mechanisms of increase in pulmonary vascular resistance. The first - a spasm of pulmonary arterioles in response to an alveolar hypoxia (reduction of partial pressure of O2 in an alveolar air). The second - reduction of total area of cross-section of a pulmonary vascular bed as a result of thrombosis or an embolism of pulmonary vessels, proliferation of smooth muscle cells in their wall, destructions of pulmonary capillaries at fibrosis or destruction of interalveolar partitions.
In response to growth of pulmonary vascular resistance or pulmonary pressure increases, but normal cordial emission remains, or cordial emission decreases, but normal pulmonary pressure remains.
Treatment of Disturbance of pulmonary blood circulation:
Treatment of the basic disease which caused disturbance of a pulmonary krovobrashcheniye is carried out.
